ClinVar Miner

Submissions for variant NM_175914.5(HNF4A):c.670+7C>T

gnomAD frequency: 0.00005  dbSNP: rs376544046
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Total submissions: 6
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Submitter RCV SCV Clinical significance Condition Last evaluated Review status Method Comment
ClinGen Monogenic Diabetes Variant Curation Expert Panel RCV005054209 SCV005687813 uncertain significance Monogenic diabetes 2025-01-29 reviewed by expert panel curation The c.670+7C>T variant in the HNF4 homeobox A gene, HNF4A, is a single nucleotide variant within intron 6 of NM_175914.5. The computational splicing predictor SpliceAI gives a score of 0.00 for donor loss, suggesting that the variant has no impact on splicing (BP4). This variant has a Popmax Filtering allele frequency in gnomAD 2.1.1 of 0.00002141 which is lower than the MDEP threshold for BS1 (0.000033) and greater than the MDEP threshold for PM2 (0.000003). This variant was identified in an individual with a clinical history highly specific for HNF4A-MODY (MODY probability calculator result >50% and negative genetic testing for HNF1A) (PP4; internal lab contributor).In summary, c.670+7C>T meets the criteria to be classified as a variant of uncertain significance for monogenic diabetes. ACMG/AMP criteria applied, as specified by the ClinGen MDEP (specification version 2.0.0, approved 10/11/2023): BP4, PP4.
Labcorp Genetics (formerly Invitae), Labcorp RCV005091185 SCV005731286 likely benign not provided 2024-09-29 criteria provided, single submitter clinical testing
Fulgent Genetics, Fulgent Genetics RCV005027601 SCV005656981 likely benign Maturity-onset diabetes of the young type 1; Type 2 diabetes mellitus; Fanconi renotubular syndrome 4 with maturity-onset diabetes of the young 2024-05-24 criteria provided, single submitter clinical testing
Clinical Genomics, Uppaluri K&H Personalized Medicine Clinic RCV003148767 SCV003804765 benign Maturity-onset diabetes of the young criteria provided, single submitter research Potent mutations in HNF4A are associated with poor insulin secretion in response to hyperglycemia. Associated with MODY1. Patients initially respond well to sulfonylureas but eventually become insulin dependent. However, more evidence is required to ascertain the role of this particular variant rs376544046 in MODY, yet.
Athena Diagnostics RCV000518627 SCV000613658 likely benign not specified 2017-06-05 criteria provided, single submitter clinical testing
PreventionGenetics, part of Exact Sciences RCV003905300 SCV004722658 likely benign HNF4A-related disorder 2019-03-04 no assertion criteria provided clinical testing This variant is classified as likely benign based on ACMG/AMP sequence variant interpretation guidelines (Richards et al. 2015 PMID: 25741868, with internal and published modifications).

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