Total submissions: 5
| Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
|---|---|---|---|---|---|---|---|---|
| Department of Pathology and Laboratory Medicine, |
RCV005397036 | SCV006053513 | uncertain significance | Spinocerebellar ataxia type 25; Combined oxidative phosphorylation defect type 13; Autosomal recessive nonsyndromic hearing loss 70 | 2021-07-30 | criteria provided, single submitter | research | |
| Ce |
RCV001840873 | SCV005433508 | uncertain significance | not provided | 2024-09-01 | criteria provided, single submitter | clinical testing | |
| Ambry Genetics | RCV004041051 | SCV005008583 | uncertain significance | Inborn genetic diseases | 2024-01-19 | criteria provided, single submitter | clinical testing | The c.40C>T (p.R14W) alteration is located in exon 1 (coding exon 1) of the PNPT1 gene. This alteration results from a C to T substitution at nucleotide position 40, causing the arginine (R) at amino acid position 14 to be replaced by a tryptophan (W). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |
| Labcorp Genetics |
RCV001840873 | SCV002474026 | likely benign | not provided | 2024-12-28 | criteria provided, single submitter | clinical testing | |
| Gene |
RCV001840873 | SCV002099641 | uncertain significance | not provided | 2022-02-28 | criteria provided, single submitter | clinical testing | In silico analysis supports that this missense variant does not alter protein structure/function; Has not been previously published as pathogenic or benign to our knowledge |