ClinVar Miner

Submissions for variant NM_000527.5(LDLR):c.648dup (p.Asp217Ter)

dbSNP: rs875989903
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Total submissions: 8
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Submitter RCV SCV Clinical significance Condition Last evaluated Review status Method Comment
Ambry Genetics RCV004992087 SCV005609304 pathogenic Cardiovascular phenotype 2024-10-05 criteria provided, single submitter clinical testing The c.648dupT pathogenic mutation, located in coding exon 4 of the LDLR gene, results from a duplication of T at nucleotide position 648, causing a translational frameshift with a predicted alternate stop codon (p.D217*). This variant has been reported in association with familial hypercholesterolemia (FH) (Defesche JC et al. J Clin Lipidol, 2017 Sep;11:1338-1346.e7; Guardamagna O et al. J Pediatr, 2009 Aug;155:199-204.e2; Di Taranto MD et al. Clin Genet, 2021 Nov;100:529-541). This variant is considered to be rare based on population cohorts in the Genome Aggregation Database (gnomAD). In addition to the clinical data presented in the literature, this alteration is expected to result in loss of function by premature protein truncation or nonsense-mediated mRNA decay. As such, this alteration is interpreted as a disease-causing mutation.
Laboratory of molecular diagnosis of dyslipidemias, Università egli studi di Napoli Federico II RCV000211601 SCV001653605 pathogenic Hypercholesterolemia, familial, 1 2021-05-24 criteria provided, single submitter clinical testing
Labcorp Genetics (formerly Invitae), Labcorp RCV001389664 SCV001591109 pathogenic Familial hypercholesterolemia 2023-07-14 criteria provided, single submitter clinical testing This premature translational stop signal has been observed in individual(s) with hypercholesterolemia (PMID: 19446849). This variant is not present in population databases (gnomAD no frequency). This sequence change creates a premature translational stop signal (p.Asp217*) in the LDLR gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in LDLR are known to be pathogenic (PMID: 20809525, 28645073). ClinVar contains an entry for this variant (Variation ID: 226328). For these reasons, this variant has been classified as Pathogenic.
Molecular Diagnostic Laboratory for Inherited Cardiovascular Disease, Montreal Heart Institute RCV001256904 SCV001433409 pathogenic not provided 2019-02-26 criteria provided, single submitter clinical testing
U4M - Lille University & CHRU Lille, Université de Lille - CHRU de Lille RCV000211601 SCV000583713 pathogenic Hypercholesterolemia, familial, 1 2017-03-30 criteria provided, single submitter clinical testing
Robarts Research Institute, Western University RCV000211601 SCV000484804 likely pathogenic Hypercholesterolemia, familial, 1 criteria provided, single submitter clinical testing
LDLR-LOVD, British Heart Foundation RCV000211601 SCV000294849 pathogenic Hypercholesterolemia, familial, 1 2016-03-25 criteria provided, single submitter literature only
Cardiovascular Genetics Laboratory, PathWest Laboratory Medicine WA - Fiona Stanley Hospital RCV000211601 SCV000268573 pathogenic Hypercholesterolemia, familial, 1 2014-08-14 no assertion criteria provided clinical testing

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