Total submissions: 13
| Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
|---|---|---|---|---|---|---|---|---|
| All of Us Research Program, |
RCV001842412 | SCV005424025 | likely benign | Cardiac arrhythmia | 2024-09-13 | criteria provided, single submitter | clinical testing | |
| Ambry Genetics | RCV002415517 | SCV002677664 | likely benign | Cardiovascular phenotype | 2021-01-30 | criteria provided, single submitter | clinical testing | This alteration is classified as likely benign based on a combination of the following: seen in unaffected individuals, population frequency, intact protein function, lack of segregation with disease, co-occurrence, RNA analysis, in silico models, amino acid conservation, lack of disease association in case-control studies, and/or the mechanism of disease or impacted region is inconsistent with a known cause of pathogenicity. |
| Molecular Diagnostic Laboratory for Inherited Cardiovascular Disease, |
RCV000987245 | SCV001433342 | uncertain significance | Brugada syndrome 1 | 2019-10-22 | criteria provided, single submitter | clinical testing | |
| Color Diagnostics, |
RCV001842412 | SCV001354424 | likely benign | Cardiac arrhythmia | 2019-03-07 | criteria provided, single submitter | clinical testing | |
| Mendelics | RCV000987245 | SCV001136495 | likely benign | Brugada syndrome 1 | 2019-05-28 | criteria provided, single submitter | clinical testing | |
| Athena Diagnostics | RCV000713149 | SCV000843728 | likely benign | not provided | 2017-11-14 | criteria provided, single submitter | clinical testing | |
| Labcorp Genetics |
RCV000713149 | SCV000291835 | likely benign | not provided | 2025-01-14 | criteria provided, single submitter | clinical testing | |
| Gene |
RCV000713149 | SCV000235314 | likely benign | not provided | 2020-12-22 | criteria provided, single submitter | clinical testing | Functional studies in mammalian cells showed that R27H causes a shift in mid-activation potential and slower channel inactivation at a less depolarized membrane potential, however R27H behaved similarly to wild type when expressed in frog oocytes (Gutter et al., 2013).; This variant is associated with the following publications: (PMID: 19716085, 23805106, 22984773, 26538325, 22840528, 11901046, 29247119, 27435932, 29728395, 30662450, 30291343) |
| Clinical Genetics DNA and cytogenetics Diagnostics Lab, |
RCV000713149 | SCV001972966 | uncertain significance | not provided | no assertion criteria provided | clinical testing | ||
| Joint Genome Diagnostic Labs from Nijmegen and Maastricht, |
RCV000713149 | SCV001953334 | uncertain significance | not provided | no assertion criteria provided | clinical testing | ||
| Stanford Center for Inherited Cardiovascular Disease, |
RCV000713149 | SCV000280484 | likely benign | not provided | 2016-12-29 | no assertion criteria provided | provider interpretation | update: reclassified variant to likely benign because of presence in 0.2% of alleles from Latinos in ExAC. |
| Forensic Genetics Laboratory, |
RCV000234990 | SCV000263114 | pathogenic | Death in infancy | 2015-03-27 | no assertion criteria provided | clinical testing | |
| Cardiovascular Biomedical Research Unit, |
RCV000058846 | SCV000090366 | not provided | Congenital long QT syndrome | no assertion provided | literature only | This variant has been reported as associated with Long QT syndrome in the following publications (PMID:11901046;PMID:19716085). This is a literature report, and does not necessarily reflect the clinical interpretation of the Imperial College / Royal Brompton Cardiovascular Genetics laboratory. |